Dilated proximal bowel → decompressed distal bowel. Identifies the site of obstruction. Distinguishes SBO from ileus (no transition point in ileus).
Air-fluid levels
Multiple, differential levels on upright film. Stepladder pattern classic for SBO.
Small bowel feces sign
Particulate matter in dilated SB near transition point. Suggests prolonged obstruction with bacterial overgrowth.
Closed-loop obstruction
U-shaped or C-shaped dilated loop with 2 transition points converging. HIGH risk of strangulation → surgical emergency.
Pneumatosis intestinalis
Air within bowel wall = ischemia/necrosis. Requires urgent surgery.
Portal venous gas
Air in portal system = bowel necrosis. Extremely ominous sign. Emergency laparotomy.
Mesenteric haziness / stranding
Suggests venous congestion or early ischemia. Correlate with lactate.
Whirl sign
Swirling of mesentery and bowel around a point = volvulus or internal hernia.
Signs of Strangulation on CT (GO TO OR): Closed-loop obstruction, pneumatosis intestinalis, portal venous gas, mesenteric haziness with non-enhancing bowel wall, free fluid, and whirl sign. Any of these = do NOT attempt conservative management.
Conservative vs Operative Management
Criteria
Conservative (Trial of Non-Op)
Operative (Go to OR)
Obstruction type
Partial SBO (contrast passes on CT)
Complete SBO, any LBO with obstruction
Etiology
Adhesive SBO (no virgin abdomen)
Hernia, closed-loop, volvulus, tumor
Clinical status
Stable, no peritonitis, tolerating NGT
Peritonitis, sepsis, hemodynamic instability
Labs
Normal lactate, normal WBC
Elevated lactate, leukocytosis, acidosis
Imaging
No closed-loop, no pneumatosis, no portal gas
Any sign of strangulation or ischemia
Time frame
Resolution expected within 48–72h
Failure to improve after 48–72h of conservative Rx
Workup
Workup
CT abdomen/pelvis with IV contrast, gold standard. Look for transition point, closed-loop, pneumatosis, portal venous gas
CBC, BMP, lactate, lipase, type and screen
Upright CXR if free air suspected (perforation)
AXR: stepladder air-fluid levels (SBO), dilated colon >6 cm or cecum >9–12 cm (LBO)
Management
Conservative (Partial SBO)
NGT decompression, NPO, IVF, serial exams q4–8h
Gastrografin (diagnostic + therapeutic: reaches colon on 24h film = resolving)
Surgery Indications
Complete obstruction, strangulation, closed-loop, failure to resolve 48–72h, incarcerated hernia
๐ Updated Practice: Old teaching: mandatory nasogastric tube (NGT) decompression for all small bowel obstruction. Current practice: NGT is indicated for patients with significant vomiting, distension, or complete obstruction. Mild partial SBO with minimal symptoms can be managed with bowel rest, IV fluids, and observation without NGT. Water-soluble contrast (Gastrografin) challenge at 24-48h can be both diagnostic and therapeutic, appearance of contrast in the colon predicts resolution without surgery.
Gastrografin Challenge Protocol
Step
Action
Details
1
Confirm appropriateness
Partial adhesive SBO, no signs of strangulation, no complete obstruction, no peritonitis
2
Administer Gastrografin
100 mL water-soluble contrast via NGT (or PO if no NGT). Clamp NGT ×2h after administration
3
Abdominal X-ray at 8–24h
Check if contrast has reached the colon/cecum
4a
Contrast in colon = RESOLVING
Advance diet. High negative predictive value for need of surgery (~98%). Defined by Defined, Abbas et al. meta-analysis, 2014
4b
Contrast NOT in colon by 24–48h
Likely requires operative intervention. Surgical consult if not already involved
Why Gastrografin works therapeutically: Hyperosmolar contrast draws fluid into bowel lumen → reduces bowel wall edema → promotes peristalsis past partial obstruction. Do NOT use barium (if perforation occurs, barium peritonitis is fatal; Gastrografin is water-soluble and absorbed).
LBO-Specific Management
Etiology
Acute Management
Definitive Treatment
Colorectal cancer
Colonic stent (bridge to surgery) or diverting colostomy
Oncologic resection with primary anastomosis or Hartmann procedure
Sigmoid volvulus
Endoscopic decompression + rectal tube
Interval sigmoid resection (recurrence rate >50% without surgery)
Cecal volvulus
Surgery (endoscopy does NOT work)
Right hemicolectomy (cecopexy has high recurrence)
Pseudo-obstruction (Ogilvie)
Neostigmine 2 mg IV (monitor for bradycardia). Colonoscopic decompression if fails
Correct underlying cause (post-op, electrolytes, medications)
Cecal diameter >12 cm = perforation risk. If cecum >9–12 cm on imaging in LBO or Ogilvie syndrome, urgent decompression is needed regardless of etiology.
Medications
Medications
Drug
Role
NS / LR
Aggressive volume resuscitation
Gastrografin
100mL via NGT (diagnostic + therapeutic)
Ondansetron
Antiemetic 4mg IV q6h
Pip-tazo / Cefepime + Metro
If strangulation/perforation suspected
On Rounds
SBO vs ileus?
SBO = mechanical (transition point on CT, dilated proximal, decompressed distal). Ileus = functional (diffuse dilation, no transition point). Ileus: post-op, opioids, hypoK, peritonitis.
Why does scope work for sigmoid but not cecal volvulus?
Sigmoid twists at accessible location → sigmoidoscopy can pass tube past twist. Cecal volvulus = cecum twists on mesentery, too proximal for scope. Cecal = surgery (cecopexy or right hemicolectomy).
What is a closed-loop obstruction and why is it dangerous?
A closed-loop obstruction occurs when a segment of bowel is obstructed at two points (e.g., adhesive band trapping a loop). The trapped segment cannot decompress proximally or distally → rapidly increasing intraluminal pressure → venous congestion → arterial compromise → ischemia and necrosis. CT shows U-shaped or C-shaped dilated loop with two adjacent transition points.
What does the Gastrografin challenge tell you, and when should you NOT use it?
Gastrografin in colon by 8–24h = SBO is resolving (98% negative predictive value for surgery). Also therapeutic, hyperosmolar contrast reduces bowel wall edema and promotes peristalsis. Do NOT use if: complete obstruction, signs of strangulation, peritonitis, or suspected perforation. Never use barium (barium peritonitis is fatal if perforation occurs). Abbas et al., Ann Surg, 2014
Name 3 CT findings that indicate bowel ischemia/strangulation and mandate surgery.
(1) Pneumatosis intestinalis, air within bowel wall = necrosis. (2) Portal venous gas, air in portal system, extremely ominous. (3) Non-enhancing bowel wall on IV contrast CT, indicates loss of blood supply. Other concerning signs: closed-loop, mesenteric haziness/free fluid, whirl sign.
What is the #1 cause of LBO and how do you manage malignant LBO acutely?
Colorectal cancer is the #1 cause of LBO. Acute management: colonic stent as a bridge to surgery (allows bowel decompression, optimization of nutritional status, and staging before elective resection) or diverting loop colostomy if stent not feasible. Definitive: oncologic resection. Emergent surgery for LBO carries higher morbidity/mortality vs elective resection after stent decompression.
How do you differentiate SBO from Ogilvie syndrome (acute colonic pseudo-obstruction)?
Ogilvie = massive colonic dilation WITHOUT mechanical obstruction. CT shows dilated colon (often >10 cm) with no transition point and no obstructing lesion. Typically post-operative, critically ill, or electrolyte deranged patients. Treatment: correct underlying cause, neostigmine 2 mg IV (monitor for bradycardia, have atropine ready), or colonoscopic decompression. Surgery if cecum >12 cm or perforation. Ponec et al., NEJM, 1999
A patient with adhesive SBO has been managed conservatively for 72 hours with no improvement. What do you do?
Failure to resolve after 48–72 hours of conservative management is an indication for surgery. Prolonged obstruction increases risk of strangulation, bacterial translocation, and aspiration. If Gastrografin was given and has NOT reached the colon by 48h, this further supports operative intervention. Consult surgery early, delayed surgical intervention in SBO is associated with increased morbidity.
Clinical Examples
📋 Case 1, Adhesive SBO Managed Conservatively
Patient: 54F with history of open appendectomy (20 years ago) and prior cesarean section. Presents with 24 hours of crampy abdominal pain, nausea, bilious vomiting ×4, and obstipation. No prior SBO episodes.
Exam: Abdomen distended, tympanitic, diffusely tender without rebound or guarding. High-pitched bowel sounds. No hernias on exam. Temp 37.1, HR 98, BP 110/70.
CT abdomen/pelvis: Dilated small bowel loops up to 4.5 cm with a transition point in the RLQ at a band adhesion. Decompressed distal ileum and colon. No closed-loop, no pneumatosis, no portal venous gas. Small amount of contrast passes the transition point (partial SBO).
Management:
Conservative: NGT placed (high output, 800 mL in first 4h), NPO, aggressive IVF with LR, K+ repletion
Gastrografin challenge: 100 mL via NGT at 24h. AXR at 8h shows contrast reaching cecum → resolving
NGT output declining. Clears by 36h. NGT removed
Diet advanced: clears → low-residue → regular. Passing flatus and stool by 48h
Discharged day 3 with dietary counseling and return precautions
Key lesson: Partial adhesive SBO without strangulation signs is the ideal candidate for conservative management. Gastrografin reaching the colon by 8–24h has a 98% negative predictive value for need of surgery.
📋 Case 2, Strangulated SBO Requiring OR
Patient: 68M with history of multiple prior abdominal surgeries (cholecystectomy, ventral hernia repair with mesh). Presents with acute-onset severe periumbilical pain ×8 hours, now constant and worsening. Multiple episodes of non-bilious emesis.
Exam: Distended, rigid abdomen with involuntary guarding and rebound tenderness. Absent bowel sounds. Temp 38.9, HR 122, BP 88/54, lactate 6.8.
CT abdomen/pelvis:Closed-loop obstruction in mid-abdomen with C-shaped dilated loop, two adjacent transition points, pneumatosis intestinalis in the affected segment, mesenteric haziness, and small-volume free fluid. No portal venous gas.
Emergency surgery: NO conservative trial, closed-loop + pneumatosis = strangulation. Taken to OR within 2 hours of arrival
Intra-op: Dense adhesive band causing closed-loop. 60 cm of non-viable small bowel (dusky, no peristalsis, no Doppler signal). Small bowel resection with primary anastomosis
Post-op ICU: continued antibiotics, serial lactate (normalized by 12h), nutrition via NG feeds on POD 3
Discharged POD 7 tolerating regular diet
Key lesson: Peritonitis + hemodynamic instability + closed-loop + pneumatosis = NO role for conservative management. These patients need immediate operative intervention. Elevated lactate >4 with leukocytosis strongly suggests bowel ischemia.
📋 Case 3, Malignant LBO with Colonic Stent
Patient: 73M with 3-week history of progressive constipation, abdominal distension, and colicky pain. No BM ×5 days. No prior abdominal surgery. 15 lb weight loss over 2 months.
Exam: Markedly distended abdomen, tympanitic. Mild diffuse tenderness, no peritonitis. Empty rectal vault on DRE. Temp 37.2, HR 90, BP 135/80.
Labs: WBC 11, lactate 1.4, Hgb 9.2 (microcytic, iron deficiency), CEA 28 (elevated).
CT abdomen/pelvis: Dilated colon (cecum 10 cm) with transition point at splenic flexure, circumferential mass causing near-complete LBO. No perforation. Multiple hepatic lesions concerning for metastatic disease. Ileocecal valve competent (no decompression into small bowel).
Management:
Acute decompression: Interventional GI places a self-expanding metal stent (SEMS) across the obstructing lesion via colonoscopy, “bridge to surgery”
Patient begins passing flatus and stool within 12h. Cecal dilation resolves on repeat AXR
MDT discussion: Given metastatic disease, neoadjuvant FOLFOX + bevacizumab started. Elective left hemicolectomy planned after 3–4 cycles if response to chemo
Stent allows nutritional optimization and avoidance of emergent surgery (which carries 15–20% mortality in obstructed CRC vs <5% elective)
Key lesson: Colonic stenting as a bridge to surgery in malignant LBO allows decompression, staging, and optimization before definitive surgery. Emergent surgery for obstructing CRC has significantly higher morbidity and mortality than elective resection. CReST Collaborative, Lancet Oncol, 2022
Monitoring
Parameter
Frequency
Abdominal exam
q4–8h
NGT output
q shift
CBC, lactate
q8–12h
Summary
Summary
SBO
#1 adhesions. NGT + NPO + IVF. Surgery if strangulation/failure 48–72h.